IgG-mediated Type III allergy – food intolerance

 

Immune complexes from IgG 1-3 antibodies and food allergies trigger the Type III food allergy, where allergens are marked by IgG 1-3-antibodies as exogenous. The same process also occurs with bacteria, viruses and other harmful substances. Thus antibody immune complexes move in the lymphs and blood through the immune complex and settle in the tissue.

In order to remove the resulting immune complex, neutrophile leukocytes release aggressive proteolytic enzymes, which trigger chronic inflammatory processes.

This reaction only ends after the food in question is no longer consumed. The illnesses caused by the Type III allergy emerge some time after the food is eaten, and only begin to slowly subside after some time.

Specialist laboratories can recognise IgG 1-3 subclass antibodies and as such the presence of Type III allergies in the blood.

The IgG-mediated Type III allergy can produce diverse symptoms. The most frequent are inflammatory skin disorders, such as acne, eczema and psoriasis, as well as inflammatory intestinal and digestive disorders. It can also cause obesity, high blood pressure, arthritis and other rheumatic illnesses.

Even if IgC antibodies are often amazingly effective as healers, they are not a cure-all treatment, as food intolerance and allergies are broad and complex disorders. The aforementioned illnesses can also be by triggered by other pathologies.

These kind of immune reactions can have various causes, are still under researched and, like the relevance of the IgC antibody in inflammatory illnesses, are still the source of some controversy.

The Type III allergy often occurs in conjunction with other underlying diseases, and shares symptoms with other pathologies, therefore a precise anamnesis and differential diagnostic knowledge are vital.

Possible triggers are

  • A primary lactose or fructose intolerance
  • Reduced intestinal flora
  • Severe intestinal yeast colonisation
  • Enzyme deficiency
  • Genetic causes
  • Increased permeability of the intestinal mucosa
  • Environmental toxins
  • Severe malnutrition
  • Premature contact with foreign proteins

In recent years, the very expensive analysis of the effectiveness of lgG anti-bodies on food has been the focus of some intense criticism from the press and specialist doctors.

I think that a lack of study of the subject and too brief anamneses, misleading and substandard laboratory analyses and bad patient education have led to many misunderstandings. The Type III allergy, which is triggered by lgG 1-3, is often confused with the presence of lgG 4. lgG 4 is an opponent of IgE immunoglobulins and is actually unsuitable for diagnosing Type III food intolerance.

In addition, the majority of medical research is financed by the pharmaceutical industry, which has no interest in carrying out comprehensive nutritional analyses, a fairly new development in laboratory medicine.

A recent double blind crossover study at Zurich University showed that Crohn’s disease patients’ symptoms – specifically stool frequency, abdominal complaints, and general wellbeing – can be significantly reduced if they are kept on an ImmuPro 300 based elimination diet.
This result underpins the theory that the consumption of food, which is thought to produce specific IgG antibodies, triggers a chronic inflammation and thus has clinical relevance for chronic inflammatory illnesses such as Chron’s disease.

During my comprehensive clinical experience with chronic inflammatory skin disorders, gastrointestinal disorders, obesity, and rheumatic disorders, I have observed how the countless patients with a proven lgG antibody response to food have been rapidly cured of chronic pain or experienced a significant improvement in their subjective wellbeing, after adhering consistently to the elimination diet.

Praxis Osteopathie & Naturheilkunde Berlin

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